Knowledge Base Neuromuscular and Nervous Systems I understand sequencing for sensory testing is superficial, deep, then cortical combined, according to O’Sullivan. I am wondering if there is a particular sequence for testing dermatome for efficiency? I think varying is best for dermatome but wanted your feedback. Here is another question – how about myotomes order sequence? Start from bottom up or top down? Thank you! Hand of benediction according to Magee, it is a ulnar nerve palsy. I have also seen in other website saying it is injury to the median nerve. Which is correct? Are synergy patterns only seen in stroke? Can they be seen in PT post TBI? Warternberg syndrome is mainly ulnar nerve involvement, PT cannot add little finger according to Magee. I also saw that it can be a radial nerve compression. Which one is true? How to differentiate between nerve root and nerve lesions? What symptomatic presentations would you expect from a PT with an AA subluxation? Would a pt be able to present with isolated neurological presentations in either the LE or UE? Explain Dermatome or Myotome pattern possibilities if any? Most of the things I’ve seen have involved are UE symptoms, perhaps due to nerve root origin. I know dystonia is an involuntary twisting and repetitive movements and Huntington’s chorea with chorea meaning having involuntary movement. But are dystonia and chorea the same thing? What kind of cues we use for right and left CVA? I am confused about the clinical presentation of anterior interosseous nerve syndrome and froment’s sign. In Brown-Sequard syndrome, if a person has right side hemisection, which side of anterolateral spinothalamic pathway is affected? According to Sillivan anterolateral spinothalamic pathway enters from dorsal root and crosses over immediately and ascends. If right side is cut then nerve fibers from right dorsal root is cut as well as right anterolateral spinothalamic pathway is cut. Are both sides anterolateral spinothalamic tracts affected? If spinal nerve roots exit above in the cervical region and below in the thoracic and lumbar region, would a disc bulge that is posterolateral at L4-L5 affect the L4 nerve root or L5? And why? In O’Sullivan, under multiple sclerosis, treatment for spasticity includes maintained stretch held for 30 mins to 3 hrs which can decrease stretch reflex activity. Won’t that cause fatigue and in turn results in relapsing of the condition? When we look for dermatomes in the body which should we remember – principal dermatome or sensory dermatome, and what is the difference between the two? (Physical therapy course manual book by IER?) In SCI, e.g. in Anterior cord syndrome will it be bilateral LMN paralysis at the level of lesion and bilateral UMN paralysis below the level of lesion? In CTSIB what is the difference between sensory selection and somatosensory? My question is about the pupillary reflex. When you use a pen in the (R) eye, which side of the eye will constrict? Is there an easy way to remember the PNF patterns? Example: which ones help with ROM, strengthening, stability, etc.? In the O’Sullivan chapter 5, it discusses measures of motor learning (pgs 189-192). It seems like the definitions of “adaptability” and “resistance to contextual change” are the exact same thing. Can you explain the difference? Can you please tell me the correct sequence for initial and later management of Lt Hemianopsia i.e. initially for left do you place items on the left as compensation and later move it to midline and then right or do you place it on the Rt since you want to encourage patient to look away from affected side? I am having doubts with ULTT 1 and 2, as both suggest 110 and 10 degree of shoulder abduction respectively, but on pg. 188 Magee, it clearly states for ULTT 2, that shoulder is more than 10′ of abduction. Am I right or I’m getting wrong somewhere? I want to know the pros and cons in the child who is habitual in doing W sitting in goal setting as PT. Does that involve a functional tenodesis effect in trunk stabilization? What is the name of the book I can find reference to this? Which Dermatome will cause paresthesias in Groin, medial thigh to knee? S1, S2, S3 or S4? What kind of deviation in the face between the trigeminal and facial nerve palsy I can differentiate? Can I have pictures of this too? If functional reach test is a test used to measure risk of fall, could the scores also be caused by merely muscle tightness causing limiting the forward reach due to tight muscles? If we are assessing a patient with a vestibular dysfunction, and we perform the hall-pike dix test and the patient has nystagmus present on both the L and R that is unidirectional, is it a peripheral dysfunction? If they had nystagmus that was bi-directional at both the L or R would this be central? I am having trouble deciphering if peripheral is strictially positional and both those scenarios would make the answer peripheral where as central the nystagmus would not be altered. Why is the leg more affected than the arm in anterior spinal cord injury patients? Concerning Obligatory Synergy Patterns, I’ve come across a couple different questions that ask what muscles or action would be the most limited based on a “typical pattern of spasticity” but wouldn’t it dependent if they were experiencing a flexion or extension synergy pattern? (question edited) Can you please clarify special test for meningeal irritation: KERNIG’s SIGN vs BRUDZINSKI’s SIGN….Sullivan 2017 has a different way of explaining compared to other sources on you tube.
I understand sequencing for sensory testing is superficial, deep, then cortical combined, according to O’Sullivan. I am wondering if there is a particular sequence for testing dermatome for efficiency? I think varying is best for dermatome but wanted your feedback. Here is another question – how about myotomes order sequence? Start from bottom up or top down? Thank you!
Hand of benediction according to Magee, it is a ulnar nerve palsy. I have also seen in other website saying it is injury to the median nerve. Which is correct?
Warternberg syndrome is mainly ulnar nerve involvement, PT cannot add little finger according to Magee. I also saw that it can be a radial nerve compression. Which one is true?
What symptomatic presentations would you expect from a PT with an AA subluxation? Would a pt be able to present with isolated neurological presentations in either the LE or UE? Explain Dermatome or Myotome pattern possibilities if any? Most of the things I’ve seen have involved are UE symptoms, perhaps due to nerve root origin.
I know dystonia is an involuntary twisting and repetitive movements and Huntington’s chorea with chorea meaning having involuntary movement. But are dystonia and chorea the same thing?
I am confused about the clinical presentation of anterior interosseous nerve syndrome and froment’s sign.
In Brown-Sequard syndrome, if a person has right side hemisection, which side of anterolateral spinothalamic pathway is affected? According to Sillivan anterolateral spinothalamic pathway enters from dorsal root and crosses over immediately and ascends. If right side is cut then nerve fibers from right dorsal root is cut as well as right anterolateral spinothalamic pathway is cut. Are both sides anterolateral spinothalamic tracts affected?
If spinal nerve roots exit above in the cervical region and below in the thoracic and lumbar region, would a disc bulge that is posterolateral at L4-L5 affect the L4 nerve root or L5? And why?
In O’Sullivan, under multiple sclerosis, treatment for spasticity includes maintained stretch held for 30 mins to 3 hrs which can decrease stretch reflex activity. Won’t that cause fatigue and in turn results in relapsing of the condition?
When we look for dermatomes in the body which should we remember – principal dermatome or sensory dermatome, and what is the difference between the two? (Physical therapy course manual book by IER?)
In SCI, e.g. in Anterior cord syndrome will it be bilateral LMN paralysis at the level of lesion and bilateral UMN paralysis below the level of lesion?
My question is about the pupillary reflex. When you use a pen in the (R) eye, which side of the eye will constrict?
Is there an easy way to remember the PNF patterns? Example: which ones help with ROM, strengthening, stability, etc.?
In the O’Sullivan chapter 5, it discusses measures of motor learning (pgs 189-192). It seems like the definitions of “adaptability” and “resistance to contextual change” are the exact same thing. Can you explain the difference?
Can you please tell me the correct sequence for initial and later management of Lt Hemianopsia i.e. initially for left do you place items on the left as compensation and later move it to midline and then right or do you place it on the Rt since you want to encourage patient to look away from affected side?
I am having doubts with ULTT 1 and 2, as both suggest 110 and 10 degree of shoulder abduction respectively, but on pg. 188 Magee, it clearly states for ULTT 2, that shoulder is more than 10′ of abduction. Am I right or I’m getting wrong somewhere?
I want to know the pros and cons in the child who is habitual in doing W sitting in goal setting as PT. Does that involve a functional tenodesis effect in trunk stabilization? What is the name of the book I can find reference to this?
What kind of deviation in the face between the trigeminal and facial nerve palsy I can differentiate? Can I have pictures of this too?
If functional reach test is a test used to measure risk of fall, could the scores also be caused by merely muscle tightness causing limiting the forward reach due to tight muscles?
If we are assessing a patient with a vestibular dysfunction, and we perform the hall-pike dix test and the patient has nystagmus present on both the L and R that is unidirectional, is it a peripheral dysfunction? If they had nystagmus that was bi-directional at both the L or R would this be central? I am having trouble deciphering if peripheral is strictially positional and both those scenarios would make the answer peripheral where as central the nystagmus would not be altered.
Concerning Obligatory Synergy Patterns, I’ve come across a couple different questions that ask what muscles or action would be the most limited based on a “typical pattern of spasticity” but wouldn’t it dependent if they were experiencing a flexion or extension synergy pattern? (question edited)
Can you please clarify special test for meningeal irritation: KERNIG’s SIGN vs BRUDZINSKI’s SIGN….Sullivan 2017 has a different way of explaining compared to other sources on you tube.